
MOTS-c
Synthetic mitochondrial-derived peptide supplied for in-vitro metabolic research.
Every lot of MOTS-c is analysed against the specification below before any vial is released.
| Parameter | Result | Method | |
|---|---|---|---|
| Appearance | White lyophilised powder | Visual | White lyophilised powder |
| Purity | ≥ 99.00% | HPLC | ≥ 95% |
| Identity | Confirmed | HPLC / MS | Meets specification |
| Heavy metals | Not detected | ICP-MS | Pass |
| Sterility | No growth | PCR | Pass |
| Endotoxin | Within limit | USP <85> | Report |
Analysis is carried out on every lot before release. If you are supplying material onward, run your own confirmatory testing on arrival — our work supports yours, it does not replace it.
- Presentation
- 10 mg / vial
- Molecular formula
- C101H152N28O22S2
- Molecular weight
- 2174.6 g/mol
- CAS
- 1627580-64-6
- Grade
- Research use only (RUO)
You'll sign in at checkout — free, and takes a moment.
Buy 2, get the 3rd 60% off. Buy 4, get 2 free — applied automatically at checkout. Or take 20% off with code PEAKSALE.
For laboratory and research purposes only. Not for human or animal consumption.
Peer-reviewed research on MOTS-c, indexed in PubMed. Listed so you can read the primary sources directly.
- Front PhysiolMitochondria-derived peptide MOTS-c restores mitochondrial respiration in type 2 diabetic heart
Pham T, Taberner A, Hickey A et al.
- Antioxidants (Basel)The Mitochondrial-Derived Peptide MOTS-c Alleviates Radiation Pneumonitis via an Nrf2-Dependent Mechanism
Zhang Y, Huang J, Zhang Y et al.
- Adv Sci (Weinh)Mitochondrial-Derived Peptide MOTS-c Suppresses Ovarian Cancer Progression by Attenuating USP7-Mediated LARS1 Deubiquitination
Yin Y, Li Y, Ma B et al.
- J Transl MedMitochondria-derived peptide MOTS-c: effects and mechanisms related to stress, metabolism and aging
Wan W, Zhang L, Lin Y et al.
- Front Endocrinol (Lausanne)MOTS-c: A promising mitochondrial-derived peptide for therapeutic exploitation
Zheng Y, Wei Z, Wang T
- Eur J PharmacolThe mitochondrial-derived peptide MOTS-c suppresses ferroptosis and alleviates acute lung injury induced by myocardial ischemia reperfusion via PPARγ signaling pathway
Lu P, Li X, Li B et al.
These papers are the published research on this molecule. They are not a claim about this product, and nothing here describes an intended use. All material is supplied for laboratory research only.
